10 free AEMT practice questions: Pulmonary Disease Processes: Sorting the Dyspneic Patient
These are real questions from the same bank the app draws from. Each one is written to the NREMT AEMT content specifications and kept inside the AEMT scope of practice. Pick an answer and you get the full rationale, including why the other three options are wrong.
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Question 1 of 10
A 27-year-old patient with asthma is sitting forward on the edge of a chair after two nebulized bronchodilator treatments. Loud wheezing is heard across all lung fields and the patient answers questions one word at a time. P 124, R 30, BP 138/84, SpO₂ 91% on room air. Which finding tells the AEMT the most about how severe the patient's airway obstruction is?
Show the answer and rationale
Correct answer · Answering questions one word at a time
This patient is having a severe asthma attack, and the one word answers tell you how bad the obstruction is. Talking takes a steady stream of air pushed past the vocal cords. Count the words a patient gets out before stopping to breathe. That count tracks how much air is really moving through the narrowed lower airways. How the chest sounds does not tell you that. A patient answering one word at a time is barely moving air. One word answers point toward respiratory failure that is about to happen. Escalate now. Call for ALS, be ready to assist ventilations with a BVM, and transport without delay.
Why the others are wrong
Loud wheezing heard across all lung fields: A loud wheeze means air is still getting through, so loudness does not measure how blocked the airways are. Wheezing only happens when air moves fast enough through narrowed airways to make sound. A chest that goes silent is the one heading toward arrest.
A heart rate of 124 after two treatments: Two nebulized bronchodilator treatments can raise the heart rate on their own, and hypoxia and the work of breathing can raise it too. With two possible causes, a rate of 124 cannot measure the airway by itself.
A respiratory rate of 30 breaths per minute: Thirty breaths a minute says nothing about how much air each breath moves. A patient taking 30 shallow breaths and a patient taking 30 effective breaths look the same on that number.
Question 2 of 10
A 74-year-old patient with a history of heart failure has worsening difficulty breathing. Fine crackles are heard at both bases, the neck veins are distended, and both ankles are swollen. The crew has laid the patient flat on the cot to carry them down the stairs, and the breathing became noticeably worse within seconds. How should the AEMT position this patient?
Show the answer and rationale
Correct answer · Upright with the legs hanging down
This patient has pulmonary edema, which is fluid backing up into the lungs from heart failure. The fine crackles at both bases, the distended neck veins, and the swollen ankles all fit heart failure. Sitting upright lets gravity pull some of the fluid away from the chest. The diaphragm also gets more room to work. Letting the legs hang down keeps blood pooled in the legs. Less blood then returns to a heart that already cannot keep up. That is why laying this patient flat made the breathing worse within seconds. Sit the patient back up, carry the patient down the stairs seated, and plan the carry around the position.
Why the others are wrong
Flat on the back with the head turned: Flat on the back is the position that just made this patient worse. Lying flat lets the blood pooled in the legs and abdomen drain back toward a heart that is already full. Turning the head does nothing about where the fluid goes.
On the left side with the knees drawn up: The left side with the knees drawn up is a comfort position for abdominal pain. This patient's problem is a heart overwhelmed with fluid, and lying on the side takes none of that load off.
Flat with the legs raised above the heart: Raising the legs above the heart sends more blood back to the center of the body. A patient in shock who needs more blood returning to the heart would get this position. This patient already has too much volume coming back, and raising the legs sends even more of it to the lungs.
Question 3 of 10
A nebulized bronchodilator works by relaxing the smooth muscle wrapped around the lower airways. Which patient's underlying problem will that action leave unchanged?
Show the answer and rationale
Correct answer · A patient whose alveoli are filled with fluid backed up from the heart
The bronchodilator leaves the patient with pulmonary edema unchanged. This patient's alveoli are filled with fluid backed up from the heart, as in left heart failure. The fluid is a pressure and volume problem, not a smooth muscle problem. There is no tight airway muscle for the bronchodilator to relax. Albuterol only acts on airway smooth muscle. Albuterol cannot pull fluid out of the alveoli or lower the pressure pushing fluid in. For this patient, CPAP comes before a nebulizer treatment. CPAP pushes fluid back into the blood vessels and takes some of the load off the heart.
Why the others are wrong
A patient whose lower airways are narrowed by bronchospasm: Bronchospasm is exactly what a nebulized bronchodilator treats. The smooth muscle is squeezing, and the drug relaxes the muscle. This patient's airway opens, and the question asks which patient's problem stays unchanged.
A patient whose airways are tightened by an allergic reaction: An allergic reaction has effects all over the body that albuterol will not fix. The airway tightening in an allergic reaction is still smooth muscle squeezing the lower airways. That muscle relaxes the same way it does in plain bronchospasm, so this patient's airway improves.
A patient wheezing during a chronic obstructive pulmonary disease flare: A COPD flare can look like the sickest patient in the group. The wheezing in a COPD flare comes from the same bronchospasm. How sick the patient is does not change what the drug acts on, so this airway still opens.
Question 4 of 10
A 58-year-old patient with difficulty breathing has a saturation of 85% on room air, so an AEMT applies an NRB at 10 L per minute. The reservoir bag empties completely every time the patient inhales. The vital signs are BP 148/88, P 112, R 26, and SpO₂ 88% on the mask. What should the AEMT do?
Show the answer and rationale
Correct answer · Raise the flow until the bag stays partly inflated through each breath
The NRB flow is too low for how hard this patient is breathing. The reservoir bag empties completely every time the patient inhales, and the SpO₂ is only 88% on the mask. The reservoir bag holds the oxygen this patient breathes in on every breath. The flow from the tank refills the bag between breaths. A bag that goes completely flat means this patient is breathing in more oxygen than 10 L per minute can refill. Once the bag is flat, the rest of that breath is room air leaking in around the mask. You do not need a different device here, only more flow. An NRB runs from 10 to 15 L per minute. Turn the flow up until the bag still has some oxygen left in it at the end of each breath.
Why the others are wrong
Leave the flow where it is, since a bag that empties shows the oxygen is being used: Some shrinking of the reservoir bag on each breath is normal. A bag that empties completely is a different finding. The flow ran out before the breath did, and this patient finished that breath on room air.
Switch to a simple face mask, which has no reservoir bag to empty: Changing devices makes sense when the one you have is failing. A simple face mask gives roughly 35 to 60 percent oxygen. That is less than the NRB was already giving this hypoxic patient.
Recheck the mask seal, since a leak at the cheeks is what empties the bag: A poor mask seal does lower the oxygen this patient gets, so checking the seal is never wasted. A leak at the cheeks lets room air in around the edges. The patient then pulls less from the bag, so the bag would empty more slowly, not faster. The seal is not what is emptying this bag.
Question 5 of 10
A patient is wheezing, and the crew is deciding whether a nebulized bronchodilator is indicated. What decides that question?
Show the answer and rationale
Correct answer · The history around the wheeze, not the wheeze by itself
The history around the wheeze decides whether a bronchodilator is indicated. A patient with known asthma or COPD and a trigger points to bronchospasm. In bronchospasm, inflamed and tight smooth muscle narrows the airways. A beta-agonist nebulizer relaxes that muscle and opens the airways back up. A history of orthopnea, paroxysmal nocturnal dyspnea, jugular venous distension, and crackles in both lung bases points to pulmonary edema from heart failure. In pulmonary edema, fluid fills the alveoli and presses on the small airways. That fluid makes the same wheezing sound. A bronchodilator does nothing to move the fluid. Get the history before you decide on a nebulizer.
Why the others are wrong
Whether the saturation has fallen below 94% on room air: A saturation below 94% on room air is a reason to give oxygen, and a low saturation tells you the patient is in trouble. The saturation measures how well oxygen gets into the blood. The saturation does not tell you what is narrowing the airway, so it cannot tell you whether to nebulize.
Whether the wheeze is heard on inspiration or on expiration: A wheeze heard only on expiration versus on both inspiration and expiration does show how far the narrowing has progressed. A wheeze on both phases still does not tell you whether asthma or heart failure caused the narrowing. The nebulizer decision depends on that difference.
The loudness of the wheeze on auscultation: A louder wheeze sounds like it should mean worse obstruction. Loudness only reflects how much turbulent air is moving through the narrowed airway. A silent chest from near-total obstruction is more dangerous than a loud wheeze. Neither loudness nor silence tells you whether bronchospasm or fluid caused the narrowing.
Question 6 of 10
An 11-year-old patient in respiratory distress has been retracting hard for twenty minutes. Over the last two minutes the retractions have eased, the patient has gone quiet, and the overall appearance has worsened. The vital signs are BP 106/62, P 138, R 16, and SpO₂ 94% on oxygen. What does that change represent?
Show the answer and rationale
Correct answer · Exhaustion, and the need to support ventilation now
This child is exhausted, and this is the point where you support ventilation. The retractions have eased and the child has gone quiet, but the overall appearance has worsened. A child who stops working to breathe while looking worse is running out of energy, not getting better. In a child, you see respiratory failure in the appearance before the pulse oximeter shows it. A saturation of 94% on oxygen does not change what the appearance is telling you. The respiratory rate has also fallen to 16. Falling effort with a worsening appearance is when you start BVM ventilation.
Why the others are wrong
A response to the oxygen that should be given more time: Oxygen does help, and giving a treatment time to work is usually wise. Here the saturation of 94% is the number that lags behind. Waiting on that number delays ventilation this child already needs.
A normal pause between episodes of increased effort: Children do tire and rally in waves, so watching for a pattern is reasonable. A quiet child who looks worse, with a respiratory rate that has fallen to 16, is not resting between episodes.
Improvement, since the work of breathing has come down: Less work of breathing is the goal of everything you are doing for this child. In a child, effort dropping while the appearance gets worse is exhaustion, never improvement.
Question 7 of 10
A 22-month-old patient has had a barking cough and noisy breathing all evening, with loud stridor at rest on arrival. Ten minutes later the stridor is much softer, the patient is limp against the caregiver, and the lips have turned cyanotic. The vital signs are BP 88/52, P 172, R 48, and SpO₂ 89% on oxygen. What does the softer stridor mean?
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Correct answer · Less air is moving, and the patient is getting worse
The softer stridor means less air is moving, and this child is getting worse. Stridor is the sound of air forcing its way past a narrowed upper airway. Making that sound takes airflow. Once the narrowing gets bad enough, the stridor fades because there is not enough air moving to make it. This child is also limp against the caregiver, with cyanotic lips and a saturation of 89% with oxygen already running. This child is heading toward respiratory arrest, not improving.
Why the others are wrong
The oxygen has taken effect and the sound will keep fading: Oxygen is the right thing to give, and oxygen does help this child. Oxygen does not shrink swelling in the upper airway. The saturation is still 89% with the oxygen running.
The swelling has come down and the patient is improving: Less noise would mean less swelling if the child also looked better. This child is limp and cyanotic. The stridor is quieter because airflow is being lost, not because the swelling went down.
The stridor has moved lower into the chest as a wheeze: Breath sounds can change as a child's condition changes. Stridor comes from a narrowed upper airway. Stridor does not move down into the chest and turn into a wheeze.
Question 8 of 10
A 60-year-old patient reports sudden onset of difficulty breathing and sharp pleuritic chest pain that began at rest, two weeks after knee replacement surgery. Lung sounds are clear and equal bilaterally. The pulse oximeter reads 88% despite high-concentration oxygen. Capnography shows a low EtCO₂ with a normal waveform shape in a tachypneic patient. Which condition is most consistent with this presentation?
Show the answer and rationale
Correct answer · Pulmonary embolism
This patient has a pulmonary embolism. Two weeks after a knee replacement, the patient had sudden difficulty breathing and sharp pleuritic chest pain that started at rest. The lung sounds are clear and equal, and the SpO₂ is 88% even on high-concentration oxygen. Not moving around after leg surgery lets a deep vein thrombosis form. That is a clot in a deep vein of the leg. A piece of that clot breaks off and gets stuck in the blood vessels of the lungs. Air still gets into the alveoli, but the blood is not getting to those alveoli. That is why the extra oxygen barely helps. The same problem explains the low EtCO₂ with a normal waveform shape. The patient is breathing fast, and the air is moving past parts of the lung that have no blood flow to drop off carbon dioxide. Clear lungs, low oxygen that does not improve on high-concentration oxygen, and a low EtCO₂ with a normal waveform all point to a pulmonary embolism.
Why the others are wrong
Asthma exacerbation: The lung sounds are clear and equal and the waveform shape is normal, so this is not asthma. Asthma gives wheezing, a long exhale, and a shark fin waveform. Asthma also tends to push the EtCO₂ up as the patient tires, not down.
Bacterial pneumonia: Pneumonia builds over a day or two with fever, a productive cough, and rhonchi or diminished sounds over one area. This difficulty breathing started suddenly, at rest. A pneumonia bad enough to drop the SpO₂ to 88% would be something you could hear with your stethoscope.
Cardiogenic pulmonary edema: happens when the left ventricle backs fluid up into the alveoli. Those patients have crackles that start at the bases, trouble breathing when lying flat, and often a history of heart failure. Fluid in the alveoli is loud, and clear, equal breath sounds rule pulmonary edema out here. The pleuritic pain and the recent surgery point to a clot instead.
Question 9 of 10
A 79-year-old patient has had three days of fever, chills, and a cough productive of thick sputum, with difficulty breathing that has worsened today. Rhonchi are heard in the right lower lung field only, and the remaining lung fields are clear. There is no jugular venous distention and no swelling of the ankles. The T is 102.4°F. The vital signs are BP 118/70, P 110, R 26, and SpO₂ 90% on room air. Which condition is most consistent with this presentation?
Show the answer and rationale
Correct answer · Bacterial pneumonia
This patient has bacterial pneumonia. Three days of fever, chills, and a cough with thick sputum fits an infection. The rhonchi are heard in the right lower lung field only, and every other field is clear. An infection fills one part of the lung, so the findings stay in that one spot. Fluid backing up from the heart and narrowing of the airways affect both lungs at once. The temperature of 102.4°F is a finding nothing else on this list explains. The SpO₂ is 90% because the alveoli in that lobe are filled with infection instead of air. Blood flowing past those alveoli never picks up oxygen.
Why the others are wrong
Cardiogenic pulmonary edema: There is no jugular venous distention and no ankle swelling, and the abnormal sounds are on one side only. A failing left heart backs fluid into both lungs, with crackles in both bases, distended neck veins, and swollen ankles. Fluid backing up from the heart also does not cause a temperature of 102.4°F.
Pulmonary embolism: Three days of slow worsening and a cough with thick sputum do not come from a clot. A clot also does not explain the fever or the rhonchi in one lobe. Pulmonary embolism brings sudden trouble breathing with clear lungs, often with chest pain on each breath and a risk factor like recent immobility, surgery, or a known clot.
Asthma exacerbation: Rhonchi in one lower lobe with every other field clear cannot come from asthma, which tightens the airways all through the chest. Asthma also does not cause fever or thick, infected sputum.
Question 10 of 10
An AEMT gave a nebulized bronchodilator to a 27-year-old patient with a history of asthma. Before the treatment, waveform capnography showed an expiratory segment that sloped upward without reaching a flat plateau, and the EtCO₂ was 52 mmHg. After the treatment, the expiratory segment rises steeply and holds a flat plateau, and the EtCO₂ is 43 mmHg. The patient now speaks in longer sentences and the accessory muscle use has decreased. What does this change most likely indicate?
Show the answer and rationale
Correct answer · Expiratory airflow has improved
The bronchodilator worked, and expiratory airflow has improved. Before the treatment, the waveform sloped upward and never reached a flat plateau. That sloped shape comes from the narrowed lower airways of asthma. Narrowed airways empty at different speeds, so exhaled gas keeps trickling to the sensor all the way through the breath out. After the treatment, the waveform rises steeply and holds a flat plateau. A flat plateau means the alveoli are emptying together again. The EtCO₂ dropped from 52 to 43 because trapped carbon dioxide is finally getting out. The patient also speaks in longer sentences and uses fewer accessory muscles, which matches the improved airflow.
Why the others are wrong
The sensor has become disconnected: A disconnected sensor makes the waveform suddenly disappear. This waveform still has a clear shape with a flat plateau and a reading of 43. Exhaled gas has to reach a connected sensor to make that waveform.
The patient is now hypoventilating: Hypoventilation makes the EtCO₂ go up. This EtCO₂ went down, from 52 to 43. The patient is also moving more air now, speaking in longer sentences with less accessory muscle use.
Air trapping has become more severe: Worsening air trapping makes the waveform slope even more and pushes the plateau further out of reach. This waveform went back toward a rectangle. Worse trapping would also push the EtCO₂ up, and this number came down.
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Airway, Respiration & Ventilation
- Advanced Airway Management and Adjuncts
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- Pulmonary Disease Processes: Sorting the Dyspneic Patient
- Suctioning and Airway Clearance
- Waveform Capnography and Ventilation Monitoring
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